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Cell Symposia: Myeloid Cells: From Development to Function and Dysfunction
June 2-4, 2021 | Shanghai, China


We understand you may be concerned about not being able to attend due to the Coronavirus COVID-19 situation. Rest assured that we will refund your registration fee, with no penalty, should you wish to cancel during the uncertainty of the outbreak.

The Elsevier conference team take the safety and well-being of their entire delegation and community very seriously. We will therefore be implementing a number of enhanced hygiene and cleaning measures, in collaboration with our conference venues, when our events take place. We will also of course, be following appropriate social distancing measures, to keep everyone safe throughout the conference.
This is coupled with our promise to refund your registration fee, with no penalty, should you wish to cancel during the uncertainty of the outbreak.

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Abstract submission deadline: February 19, 2021
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Myeloid cells – mononuclear (monocytes, macrophages, and dendritic cells (DCs)) and polymorphonuclear (neutrophils, eosinophils, basophils, and mast cells), as well as immature myeloid progenitors of both lineages -- play critical roles in immune homeostasis and inflammation. Their dysregulation has been linked to cancer, metabolic, inflammatory diseases, and degenerative diseases, making them increasingly important potential targets for therapeutic intervention.

Together we will discuss areas of long-standing and topical interest in the field of myeloid cell biology, including:

  • Ontogeny and basic function
  • Resolution of inflammation and cellular cross-talk
  • Dysregulation of myeloid cell function in the context of autoimmunity and allergy
  • The role of myeloid cells in metabolic disease and the role of intermediary metabolism in myeloid function
  • The role of myeloid cells in the control and support of tumorigenesis and tumor growth

We hope to bring together basic scientists, clinicians, and industry to discuss new findings and future challenges in investigating these sentinels of the immune system.


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